Autoimmune CKD Remission and improving Kidney Function

by Jothi Prem | Jul 14, 2023 | English, Kidney Disease

As I am doing well in stopping the progression and improving kidney function among Early-stage CKD patients due to Long-term Diabetes and high blood pressure,  I started getting calls from patients with CKD due to various auto-immune diseases like IgA Nephropathy, Membranous Nephropathy, Lupus, etc. One patient, Ritika, contacted me to get treatment for her IgA Nephropathy. She was 28 yr old and when she got diagnosed, her creatinine was 2.6 with very high blood pressure. She started taking medicines given by doctors for IgA and also for high blood pressure. Slowly every month her creatinine level increased to 3.2. She decided to get treated at my clinic for 4 weeks and was able to bring down creatinine to 2.59 in three weeks. She went back to Chhattisgarh to continue the protocol. She got a fever after reaching there while traveling and her creatinine got increased to 2.8. Then I told her to continue my protocol. After two weeks, her creatinine came down to 2.4. Now her creatinine is oscillating between 1.5. Now I am convinced that my protocol really works for those whose creatinine is less than 4 and good urine output. You can watch her testimonials below.

Another patient’s parents contacted me for their son, Mr. Tarun, age 21, suffering from IgA nephropathy. His creatinine was 1.7 and decided to get treated at an early stage and took 3 weeks of treatment. We are able to bring it down to 1.5. His protein leak was completely nil and no microscopic blood cells, which is the main symptom of IgA.

The Real Cause of CKD due to auto-immune 

By going through the following excerpts from research papers you can understand Viral infection’s role.

IgA Nephropathy: Pleiotropic impact of Epstein-Barr virus infection on immunopathogenesis and racial incidence of the disease

In analyses of sera and cells from White IgAN patients, healthy controls, and African Americans, IgAN patients exhibited substantial enrichment for IgA-expressing B cells infected with Epstein-Barr virus (EBV), leading to enhanced production of poorly galactosylated IgA1.

Systemic Epstein-Barr virus infection associated with membranous nephropathy in children

Nephrotic syndrome developed at initial presentation and was associated with MN on biopsy. The patient cleared the virus from blood, which was associated with eventual resolution of the MN

Epstein-Barr Virus and Systemic Lupus Erythematosus

Many studies have revealed a connection between SLE and EBV infection. Essentially all adult SLE patients are infected with EBV (99.5%) [24]. However, the statistical significance of this finding is reduced by the large proportion of healthy adults infected as well (95%).

Acute Epstein–Barr virus infection-associated collapsing glomerulopathy

A 21-year-old woman presenting with acute Epstein–Barr virus (EBV) infection (infectious mononucleosis) was noted to have renal involvement. She had proteinuria, leukocyturia and microscopic hematuria, and 10 days after admission became nephrotic (23 g of protein per g of creatinine). Renal biopsy revealed glomerular tuft collapse, visceral epithelial cell proliferation and vacuolization consistent with collapsing glomerulopathy. She had only transient deterioration in renal function, attributed to contrast nephropathy, but after recovery remained proteinuric.

Infection-Related Focal Segmental Glomerulosclerosis in Children

In adults, the most established FSGS causing virus is the human immune-deficiency virus, which is related to a collapsing variant of FSGS. Nevertheless, other viruses are also suspected for causing a collapsing or noncollapsing variant, for example, hepatitis B virus, parvovirus B19, and Cytomegalovirus.

Viruses and collapsing glomerulopathy: a brief critical review

Case reports and small-case series link infection with these common viruses and glomerular injury. The evidence for a pathogenic role is generally stronger for glomerulonephritis than for collapsing glomerulopathy

How My Non-toxic Treatment Works

In modern medicine all chronic diseases are treatable. I believe all chronic diseases can be reversible or put into remission by understanding the real problem at the molecular level. Genes can be turned on and Off by Diet and Lifestyle. This is my 14 years of experience in learning proven scientific non-toxic therapies using diet, Lifestyle, Nutrition, and plain herbs.

I have a small farmhouse, in Thanjavur, Tamil Nadu, which I call a Lifestyle center. My passion was healing incurable diseases. I am good at finding the pathophysiology of diseases and finding solutions by treating the root cause.

I am getting good results in stopping and reversing chronic kidney diseases caused by diabetes and high blood pressure. People with auto-immune diseases like IgA, IgG, IgM, IgE, lupus, etc…. started contacting me for treatment. Slowly I started studying research papers about these diseases and got convinced that this deadly disease can be handled using advanced enzyme therapy, plain herbs (non-ayurvedic), clinically proven diet, and Nutrition.

There are herbs that suppress the malfunctioning immune system and there are clinically proven herbs that are anti-viral and modulate the immune system to fight against viruses and lead to remission of CKD. Enzymes help in healing the damaged nephrons.

For example, in IgA nephropathy, a higher level of galactose deficient, defective IGA antibody called IgAN is produced.  IgA is triggered due to infection in the lungs, digestive tract, and tonsils in IgAN patients. The IgG antibodies consider the IgAN as a foreign body and bound to the galactose-deficient sugar side chain of the IgAN and form an antibody complex, which gets accumulated in the kidney and damages the nephrons.

I have studied enzyme therapies very deeply. There are specific enzymes good for dissolving immune complexes. Various Enzymes  help in healing nephrons, dissolving viruses and immune complexes, and dissolves viruses and fibrins. Also, enzymes that dissolves biofilms of drug-resistant bacterial infections which trigger IgA production. Once the infections are cleared, there is the possibility of IgAN remission or a low level of IgAN production. In either case, taking a maintenance dose of enzymes that dissolve the immune complex life which causes no side effects is safe for preventing relapse, as per my current understanding.

My protocol helps those who have good urine output. Once a nephron is dead, it is dead, rejuvenating new nephrons is rare. One can heal the damaged nephrons and prevent further damaging nephrons through my therapy. Depending on, how good is the kidney, diet and therapy varies.   The earlier you start the treatment, creatinine, and urea can be reduced, and can live a normal life, life long.

If you are suffering from auto-immune-related CKD and want to bring down creatinine and improve kidney function, Contact me via WhatsApp by tapping below.